Brain tissue ammonia and Alzheimer type 2 astrocytosis in human liver cirrhosis

dc.contributor.authorShuliatnikova, T. V.
dc.contributor.authorTumanskyi, V. O.
dc.contributor.authorШулятнікова, Тетяна Володимирівна
dc.contributor.authorТуманський, Валерій Олексійович
dc.date.accessioned2023-06-15T11:26:37Z
dc.date.available2023-06-15T11:26:37Z
dc.date.issued2023
dc.description.abstractHepatic encephalopathy (HE) is among key manifestations of decompensated liver cirrhosis (LC) and is typical for 30-45% of patients [1], reducing their survival rate up to 2 years. The neurotoxic effect of ammonia is central in the pathophysiology of HE [2]. In astrocytes, high ammonia concentration with glutamate are metabolized in osmotically active glutamine, accumulation of which causes astrocytes swelling and subsequent generalized brain edema [3].uk_UK
dc.identifier.citationShuliatnikova T. V. Brain tissue ammonia and Alzheimer type 2 astrocytosis in human liver cirrhosis / T. Shuliatnikova, V. Tumanskyi // Theory and practice of modern science : collection of scientific papers "SCIENTIA" with Proceedings of the V International Scientific andTheoretical Conference, May 12, 2023. Kraków, Republic of Poland. - Kraków : European Scientific Platform, 2023. - P. 161-162.uk_UK
dc.identifier.urihttps://zsmu.rosbai.com/handle/123456789/18951
dc.language.isoenuk_UK
dc.subjectтези доповідейuk_UK
dc.titleBrain tissue ammonia and Alzheimer type 2 astrocytosis in human liver cirrhosisuk_UK
dc.typeArticleuk_UK

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