Astroglial reactivity in the experimental acute hepatic encephalopathy

dc.contributor.authorShuliatnikova, T. V.
dc.contributor.authorШулятнікова, Тетяна Володимирівна
dc.date.accessioned2021-10-27T11:02:54Z
dc.date.available2021-10-27T11:02:54Z
dc.date.issued2021
dc.description.abstractLiver pathologies associated with severe functional failure in most cases are complicated by potentially reversible but still dangerous neurocognitive disorder termed acute hepatic encephalopathy (AHE) [1]. It is believed that AHE underlying mechanisms are generally focused on the ammonia neurotoxicity. Hyperammonemia mostly target brain astrocytes as they are the only cell population that contain glutamine synthetase and metabolize ammonia along the brain [2].uk_UK
dc.identifier.citationShuliatnikova T. V. Astroglial reactivity in the experimental acute hepatic encephalopathy / T. V. Shuliatnikova // Débats scientifiques et orientations prospectives du développement scientifique : collection de papiers scientifiques «ΛΌГOΣ» avec des matériaux de la II conférence scientifique et pratique international, (Vol. 2), Paris, 1er octobre 2021. - Paris-Vinnytsia : La Fedeltà & Plateforme scientifique européenne, 2021. - С. 41-42. - DOI 10.36074/logos-01.10.2021.v2.15.uk_UK
dc.identifier.urihttps://zsmu.rosbai.com/handle/123456789/15126
dc.language.isoenuk_UK
dc.titleAstroglial reactivity in the experimental acute hepatic encephalopathyuk_UK
dc.typeArticleuk_UK

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