Molecularly-biochemical and neuropsychophysiological aspects of neuroprotective activity of mexidol during chronic immobilizational stress

dc.contributor.authorChekman, I. S.
dc.contributor.authorYakovleva, I. Yu.
dc.contributor.authorBelenichev, I. F.
dc.contributor.authorGorchakova, N. A.
dc.contributor.authorBuhtiyarova, N. V.
dc.contributor.authorБєленічев, Ігор Федорович
dc.contributor.authorБухтіярова, Ніна Вікторівна
dc.date.accessioned2018-04-03T12:35:19Z
dc.date.available2018-04-03T12:35:19Z
dc.date.issued2013
dc.description.abstractAbstract: It is found out that in models of rats’ chronic stress an oral administration of Mexidol (100 mg/kg) during 10 days decreases a degree of DNA oxidative modification markers (8-OHG), proteins APH and KPH, hyperproduction of NO, and activity of NO-synthase. Also it changes an expression of early gene c-Fos and increases a concentration of Bcl-2 in neurons of paraventricular nuclei of the hypothalamus.uk_UK
dc.identifier.citationMolecularly-biochemical and Neuropsychophysiological Aspects of Neuroprotective Activity of Mexidol during Chronic Immobilizational Stress / I. S. Chekman, I. Yu. Yakovleva, I. F. Belenichev, N. A. Gorchakova, N. V. Buhtiyarova // Molecular Pharmacology. – 2013. – Issue 2. – P. 1-6.uk_UK
dc.identifier.issn0976-3856
dc.identifier.urihttps://zsmu.rosbai.com/handle/123456789/7554
dc.language.isoenuk_UK
dc.publisherUnited States : American Society for Pharmacology and Experimental Therapeuticsuk_UK
dc.titleMolecularly-biochemical and neuropsychophysiological aspects of neuroprotective activity of mexidol during chronic immobilizational stressuk_UK
dc.typeArticleuk_UK

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