Molecular and biochemical mechanisms of mitochondrial dysfunction in spontaneously hypertensive rats
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Hikari Ltd
Abstract
Objective: In conditions of hypertension, ATP synthesis can be violated that can
lead to the development of energy deficit of cardiomyocytes. One of the reasons
of such energy deficit could be mitochondrial dysfunction. Aim of this work was
study of molecular and biochemical mechanisms of mitochondrial dysfunction in
spontaneously hypertensive rats.
Description
Citation
Molecular and biochemical mechanisms of mitochondrial dysfunction in spontaneously hypertensive rats / E. A. Nahorna, I. S. Chekman, I. F. Belenichev, Yu. M. Kolesnik, A. V. Abramov, N. A. Gorchakova, S. V. Levich // Biological markers and guided therapy. – 2016. – Vol. 3, № 1. – P. 73-85.