Molecular and biochemical mechanisms of mitochondrial dysfunction in spontaneously hypertensive rats

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dc.contributor.authorNahorna, E. A.
dc.contributor.authorChekman, I. S.
dc.contributor.authorBelenichev, I. F.
dc.contributor.authorKolesnik, Yu. M.
dc.contributor.authorAbramov, A. V.
dc.contributor.authorGorchakova, N. A.
dc.contributor.authorLevich, S. V.
dc.contributor.authorНагорна, О. А.
dc.contributor.authorЧекман, І. С.
dc.contributor.authorБєленічев, Ігор Федорович
dc.contributor.authorКолесник, Юрій Михайлович
dc.contributor.authorАбрамов, Андрій Володимирович
dc.contributor.authorГорчакова, Н. А.
dc.contributor.authorЛевіч, Сергій Вадимович
dc.date.accessioned2018-10-16T11:52:28Z
dc.date.available2018-10-16T11:52:28Z
dc.date.issued2016
dc.description.abstractObjective: In conditions of hypertension, ATP synthesis can be violated that can lead to the development of energy deficit of cardiomyocytes. One of the reasons of such energy deficit could be mitochondrial dysfunction. Aim of this work was study of molecular and biochemical mechanisms of mitochondrial dysfunction in spontaneously hypertensive rats.uk_UK
dc.identifier.citationMolecular and biochemical mechanisms of mitochondrial dysfunction in spontaneously hypertensive rats / E. A. Nahorna, I. S. Chekman, I. F. Belenichev, Yu. M. Kolesnik, A. V. Abramov, N. A. Gorchakova, S. V. Levich // Biological markers and guided therapy. – 2016. – Vol. 3, № 1. – P. 73-85.uk_UK
dc.identifier.urihttps://zsmu.rosbai.com/handle/123456789/8378
dc.language.isoenuk_UK
dc.publisherHikari Ltduk_UK
dc.subjectHypertensionuk_UK
dc.subjectheat shock proteinsuk_UK
dc.subjectmitochondrial dysfunctionuk_UK
dc.titleMolecular and biochemical mechanisms of mitochondrial dysfunction in spontaneously hypertensive ratsuk_UK
dc.typeArticleuk_UK

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