Expression of HSP70 in the brain of rats during experimental cerebral ischemia modeling and on the background of neuroprotection

dc.contributor.authorBelenichev, I. F.
dc.contributor.authorPavlov, S. V.
dc.contributor.authorBukhtiayarova, N. V.
dc.contributor.authorSamura, I. B.
dc.contributor.authorEgorov, A. N.
dc.contributor.authorSemenov, D. M.
dc.contributor.authorБєленічев, Ігор Федорович
dc.contributor.authorПавлов, Сергій Васильович
dc.contributor.authorБухтіярова, Ніна Вікторівна
dc.contributor.authorСамура, Ірина Борисівна
dc.contributor.authorЄгоров, Олександр Миколайович
dc.contributor.authorСеменов, Денис Михайлович
dc.date.accessioned2018-05-21T12:10:28Z
dc.date.available2018-05-21T12:10:28Z
dc.date.issued2017
dc.description.abstractExperimental research has found that acute cerebral ischemia modeling in rats is accompanied by a significant increase in the main marker of oxidative degradation of proteins – nitrotyrosine beginning from the first day of the experiment. Concurrently there has been a decline in stress-protein HSP70 in brain tissue. Administration of Cerebrocurin for 4 days has led to a decrease in the levels of nitrotyrosine, as well as to increase in the expression of stress-responsive protein HSP70 in the acute period of cerebral ischemia.uk_UK
dc.identifier.citationExpression of HSP70 in the brain of rats during experimental cerebral ischemia modeling and on the background of neuroprotection / I. F. Belenichev, S. V. Pavlov, N. V. Bukhtiayarova, I. B. Samura, A. N. Egorov, D. M. Semenov // Biological Markers and Guided Therapy. – 2017. – Vol. 4. – P. 105 - 111.uk_UK
dc.identifier.urihttps://zsmu.rosbai.com/handle/123456789/7745
dc.language.isoenuk_UK
dc.publisherBulgaria: Hikari Ltduk_UK
dc.subjectHSP70uk_UK
dc.subjectcerebral ischemiauk_UK
dc.subjectneuroprotectionuk_UK
dc.titleExpression of HSP70 in the brain of rats during experimental cerebral ischemia modeling and on the background of neuroprotectionuk_UK
dc.typeArticleuk_UK

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